How
Mobile Phones Could Provide a Breakthrough for
Alzheimer’s
After
years of hearing that mobile phones can cause brain tumours, an
inventive new
study suggests mobile phone radiation may actually have a beneficial
biological
effect—two hours of exposure a day staved off Alzheimer’s disease in
mice.
Scientists
at
the University of South Florida studied mice that are genetically
predisposed
to develop Alzheimer’s and its accompanying memory problems. Based on
previous
research, the researchers hypothesized that radiation from phones would
accelerate progression of the disease because other types of radiation
cause
free radical damage. The team used an antenna to expose some of the
mice to
electromagnetic waves that approximated two hours of daily cell phone
use. To
the scientists’ surprise, the mice that were dosed with mobile phone
radiation
did not suffer from memory impairments as they aged—unlike their
radiation-free
counterparts. The mice exposed to phone waves retained their youthful
ability
to navigate a once familiar maze after time spent in different mazes.
The
researchers have a theory that the radiation prevented the buildup of
amyloid
plaques, the sticky protein aggregates that are found in Alzheimer’s
brains. They
suggest that their work may eventually lead to a treatment that can
halt the
disease process.
Studies
in
mice are at very early stages, of course: many avenues of treatment
that
seem promising in rodents fail to live up to their potential in humans.
But the
new research raises questions about the mobile phone industry’s claim
that its
products’ emissions are too weak to have any biological effects!
Although the link
to brain tumors remains inconclusive, the new work suggests mobile
phones may
indeed have an effect on our brains….
References:
G.W.
Arendasha, J.
Sanchez-Ramos, T. Morie, M. Mamcarz, X. Lin, M.
Runfeldt, L. Wang, G. Zhangb, Vasyl Sava, J. Tang and C. Cao (2010). Electromagnetic Field
Treatment Protects Against and Reverses Cognitive Impairment in
Alzheimer’s
Disease Mice. Journal of Alzheimer’s Disease 19(1),
191–210.